The Genetics Of Obstructive Sleep Apnea PMC

is sleep apnea genetic

While central sleep apnea does not appear to be an inherited disorder, obstructive sleep apnea can be genetic. Having a parent, sibling, or child with OSA increases a person’s here likelihood of developing OSA by about 50 percent. To understand why only OSA runs in families, it’s important to know how the two forms of sleep apnea differ.

While you can’t control the genetic predisposition to sleep apnea, you can make choices that can help you avoid developing conditions that increase your risk for obstructive sleep apnea (OSA), like obesity and high blood pressure. Studies have identified some additional factors that are linked with a heightened risk of central sleep apnea. After discussing your sleep apnea symptoms and medical history with your doctor, you will likely participate in a sleep study to receive a diagnosis. In a sleep study, your heart rate, breathing, blood oxygen levels, electrical activity, and other physiological variables are monitored overnight at a sleep center or at home. In cases where the underlying cause of central sleep apnea is never identified, the condition may be considered primary central sleep apnea or idiopathic central sleep apnea13, Dr. LoSavio says. You may be able to reduce your risk of developing central sleep apnea by caring for any underlying medical conditions you may have and talking to your doctor about alternative medications if you take opiates11.

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Your profile will connect you to sleep-improving products, education, and programs curated just for you. In order to collect more information about what these details happens while you sleep, the doctor may ask for a sleep history not just from you, but also from someone who shares a bed or household with you.

is sleep apnea genetic

Other types of PAP devices, such as bi-level positive airway pressure (BiPAP) and auto-titrating positive airway pressure (APAP), provide variability in the amount of air pressure. In OSA, a narrowing of the airway during sleep leads to breathing disruptions. In CSA, the breathing disruptions are caused by a lack of communication between the brain and the muscles involved in breathing.

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Studies suggest that variations in multiple genes, each with a small effect, combine to increase the risk of developing the condition. However, it is unclear what contribution each of these genetic changes makes to disease risk. Most of the variations have been identified in single studies, and subsequent research has not verified them.

Concurrent with increased clinical recognition of OSA and importance as a chronic disease, there have been major advances in genetic methodologies to discover genetic susceptibility loci for many conditions. For they said example, 97 validated loci have been discovered by genome wide association studies (GWAS) for obesity9. Relative to many other common, chronic disorders, the genetic understanding of OSA has lagged behind.

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