Is Back Pain A Symptom Of COVID-19? What Research Shows

back pain covid

Consequently, the actual number of patients with COVID-19 will be much higher than the estimated number, and an enormous portion of the world population will be bearing the health consequences of postacute COVID-19 syndrome. LBP was measured using the musculoskeletal his response subscale of subjective health complaints produced by Eriksen et al. Descriptive analysis was performed to compute LBP prevalence and compare the prevalence across groups. Multiple logistic analyses helped to identify the predictors of LBP for survivors of COVID-19.

back pain covid

The authors did not find any difference in the presentation of COVID-19-related CIM/CIP from other causes. However, they observed a noticeable degree of spontaneous muscle activity in patients COVID-19 patients with CIM. Although previous research has suggested that back pain may be a relatively common symptom during the initial stages of infection particularly in the Omicron era  the extent to which low back pain affects people during their recovery was less certain. If a person has lower back pain alongside other symptoms that suggest a viral infection, they should undergo testing. Long-term effects of COVID mostly occur among people who previously experienced severe symptoms of the disease.

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Despite these limitations, this study provided valuable baseline information regarding the association between SARS-CoV-2 infection and LBP. The mechanism behind the presence of myalgia or arthralgia with COVID-19 navigate to these guys infection remains poorly understood. As a close relative to COVID-19, studies on SARS-CoV-1 may provide insight into potential mechanisms of injury to the musculoskeletal system caused by COVID-19 infections.

Studies have also suggested that the presence of arthralgia may have an association with disease severity, although the evidence for this is scarce. Furthermore, in patients experiencing long-term effects of COVID-19 or a prolonged disease course, 27% report ongoing joint pain. Patients with arthralgia also reported higher pain levels and required more analgesia. Therefore, it is imperative for clinicians to recognize this when diagnosing and managing patients with a history of COVID-19 infection who present with arthralgia and to adequately discern this finding from reactive arthritis.

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Second, the data collector gathered particulars of randomly selected 600 (60 from each center) subjects, aged 18 and older, who previously tested positive for COVID-19 but have no active illness. Considering inclusion and exclusion criteria, 450 subjects were found to be eligible for this study. Finally, participants who consented to have their data collected underwent individual face-to-face interviews at their homes or workplaces; therefore, 439 case data were secured.

One case series reported that of 225 COVID-19 patients in the ICU in Spain, 12 patients were referred to the neurophysiology department for suspicion of ICUAW, and 11 cases of CIM and CIP were confirmed. This reported rate of ICUAW in ICU patients with COVID-19 is thought to be underestimated due to patient death prior to diagnosis, the delay and cancellation of non-essential studies due to the infectious nature of the disease, and limited availability of neurophysiology staff and resources. It is essential that clinicians can identify source and properly diagnose COVID-19 patients with ICUAW so that these patients may receive early rehabilitation and treatment to improve functional outcomes after recovery. Additionally, it is important to recognize that CIM has a better prognosis than CIP according to published studies and younger patients with CIM are found to have more positive outcomes and recovery of motor skills. Additionally, it is imperative to investigate the pathology, and potential mechanisms of the impact COVID-19 has on the musculoskeletal system.

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